Treatment with liraglutide was associated with 6.1% weight loss and reduced diabetes risk by nearly 80%.
In an early trial, patients lost an average of 37% of their excess weight in 4 months.
Two genetic variants are associated with heightened brain reward responses to foods high in fat and sugar.
Mothers with a high-fat diet may program their offspring for increased weight.
New data link hypothalamic gliosis with higher BMI and increased insulin resistance.
Weight, metabolic health, and overall quality of life improved 3 years after bariatric surgery.
Despite high engagement, a cell phone intervention did not increase weight loss.
Children of Latino parents with high stress levels are twice as likely to be obese.
Noninvasive brain stimulation may help decrease caloric intake and promote weight loss.
One night of sleep deprivation may be as detrimental as 6 months on a high-fat diet.
Health insurance often excludes obesity treatments, even when target BMI is included in employee wellness programs.
Stress and genetics may also play a role in the effect of food cues on some people.
Endocrinology Advisor Articles
- Effect of Sugar-Sweetened Beverages on Obesity Development
- Metformin and PCOS: Improving Menstrual Cycle and Hormone Profiles
- Denosumab May Increase BMD in Women With Primary Hyperparathyroidism
- T2D May Play a Protective Role Against Developing Aortic Diseases
- Risk Factors for Diabetes May Be Identified 20 Years Before Diagnosis
- Metformin Safe in T2D With Moderate to Severe Chronic Kidney Disease
- SGLT2 Inhibitors Show High Cardiovascular- and Renal-Protective Effects in T2D
- Type 2 Diabetes Insulin Initiation: Predictors Identified
- Two New Diabetes Medications Now Available
- Managing Acute Pain in Patients With Severe Obesity
- Risk for Congenital Malformations With Antithyroid Drugs
- Occupational Therapy May Benefit Young Adults With Diabetes
- Weight Loss May Improve Pain in Patients With Obesity
- Prior Authorizations Are Creating a Challenge for Clinicians
- Effect of Biologic DMARDs on Spinal Fracture Risk in Ankylosing Spondylitis